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CD22-targeted CAR T-cell therapy At least 50% of relapses after CD19-targeted CAR T-cell therapy have occurred because of antigen escape, which has been shown to be related to mutations in the CD19 protein that delete the binding sites used by CAR T-cell constructs.[203] Salvage after antigen escape has been documented with cell and immune therapy approaches targeting a second lymphoid antigen, CD22

The hallmark anatomic finding is fusion of the valve commissures, producing a "fish-mouth" deformity that obstructs diastolic ventricular filling

A novel cationic niosome formulation for gene delivery to the retina

However, current evidence suggests that the development of such antibodies does not significantly reduce the drugs effectiveness or cause any distinct adverse reaction profile