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NAC may be a direct source of cysteine following hydrolysis or may reduce plasma cystine through thiol- disulfide exchange, liberating endogenous cysteine

Such findings highlight that transient p21 induction favors cell survival by buying time for repair, whereas sustained activation may drive cells into senescence or confer therapy resistance depending on context

10.17533/udea.vitae.v28n1a342472 [DOI] [Google Scholar] Medicines and Healthcare products Regulatory Agency (2005)

Front Pharmacol 12:782660 Li P, Inoue Y, Miyamoto D, Adachi T, Okada S, Adachi T, Soyama A, Hidaka M, Kanetaka K, Ito S (2023) Therapeutic effect and mechanism of Daikenchuto in a model of methotrexate-induced acute small intestinal mucositis