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The peptide was formed by fusing the HIV Tat (transcriptional counter-activator) signal (YGRKKRRQRRR) with a competing peptide spanning amino acids 125 to 135 (YEMPSEEGYQD), the phosphorylation site (ser129) in -syn (siP-Syn)
Importantly, in vitro treatment with TNFi did not attenuate the cell death phenotype
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